BOOMB TRAS BOOM << Alsee salva el mundo de nuevo >> LA RECETA ALLSE 1 SHOT 1 KILL PARA EL (FAKE) INFO cobi19 (5G / SATELITE) no magufada .

[allseeyingeye]

me encontraba relajadamente meditando sobre genialidades varias





y no vais a creer lo que paso...

ME VINO UN INSIGHT DE LA FUENTE ORIJINAL PURA


NAC ACETIL CISTENIA
THE BRUTAL bichito KILLER DERROYER


0.20 SEGUNDOS DE INVESTIGACION SUPREMA


1 SHOT

1 KILL



DE HECHO LO ACABO DE TOMAR

ME HE SENTIDO BIEN IPSO FACTO A PESAR DE NO TENER NADA

Y ME HE ANIMADO A HACER EL POST

RESULTADOS PUEDE VARIAR

en mi caso me he escrito a buscarlo en 0.2 segundos, por que estaba como cansao, he cogido un poco de NAC que tenia por ahi muerto de risa, ha sido tomarlo con vitaminas
y me han entrando ganas de escribir y me noto menos cansado



ACLARACION. NO ES puñetero MAGICA

DEBERIA SER PARTE DE TODO UN PROTOCOLO CON MIL COSAS

PERO LO ABSTRAC ES DIOS

SE LO DAN A LOS VIEJOS Y PICHAN IGUAL

PERO POR QUE LES DAN MAS COSAS PA QUE PICHE




https://www.who.int/csr/disease/coronavirus_infections/MERSCov_WHO_KSA_Mission_Jun13u.pdf
SAGE Journals: Your gateway to world-class research journals

• Format: Abstract

Biochem Pharmacol. 2010 Feb 1;79(3):413-20. doi: 10.1016/j.bcp.2009.08.025. Epub 2009 Sep 2.
N-acetyl-L-cysteine (NAC) inhibits bichito replication and expression of pro-inflammatory molecules in A549 cells infected with highly pathogenic H5N1 influenza A bichito.
Geiler J1, Michaelis M, Naczk P, Leutz A, Langer K, Doerr HW, Cinatl J Jr.
Author information
pathways includ

SAGE Journals: Your gateway to world-class research journals
Abstract
The antioxidant N-acetyl-L-cysteine (NAC) had been shown to inhibit replication of seasonal human influenza A viruses. Here, the effects of NAC on bichito replication, bichito-induced pro-inflammatory responses and bichito-induced apoptosis were investigated in H5N1-infected lung epithelial (A549) cells. NAC at concentrations ranging from 5 to 15 mM reduced H5N1-induced cytopathogenic effects (CPEs), bichito-induced apoptosis and infectious viral yields 24 h post-infection. NAC also decreased the production of pro-inflammatory molecules (CXCL8, CXCL10, CCL5 and interleukin-6 (IL-6)) in H5N1-infected A549 cells and reduced monocyte migration towards supernatants of H5N1-infected A549 cells. The antiviral and anti-inflammatory mechanisms of NAC included inhibition of activation of oxidant sensitive pathways includ
SAGE Journals: Your gateway to world-class research journals

ERNATIONALJOURNALOFIMMUNOPATHOLOGYANDPHARMACOLOGYVol. 20, no. 2,349-354(2007)N-ACETYLCYSTEINESYNERGIZESWITHOSELTAMIVIRINPROTECTINGMICEFROMLETHALINFLUENZAINFECTIONA. GAROZZO, G. TEMPERA, D.UNGHERI',R.TIMPANARO and A. CASTRODepartmentofMicrobiologicalandGynaecologicalSciences, UniversityofCatania, Catania;JGlobalScientificProjectManagement, Zambon Italia Sirl., Milano, ItalyReceivedSeptember20,2006-AcceptedFebruary20,2007Manystudieshaveshownthatoxidativestressisimportantinthepathogenesisofpulmonarydamageduringinfluenzavirusinfections.Antioxidantmoleculesarethereforepotentiallyusefulagainstviralinfection.OurpreviousstudiesshowthatN-acetytcysteine(NAC)hasa protectiveeffect in amodeloflethalinfluenzainfectionin mice. NACadministrationsignificantlydecreasedthemortalityininfectedmice.FurtherstudieshavedemonstratedthatNACenhancedsurvivalincombinationwiththeantiviralagentribavirin.Inthepresentstudy,wereporttheeffectofcombinedtreatmentwithNACandOseltamivir,clinicallyused inthetreatmentandpreventionofinfluenzavirusinfection,in amurinemodeloflethalinfluenzainfection.NAC was given as a singledailydose of 1000mglKgstartingfrom4 hbeforeinfectionanduntilday4afterinfection;Oseltamivirwas given twicedailyat dose of 1mglKgldiefor 5days,startingfrom4 hbeforeinfection.End-pointevaluationwas21-days'survival.NACalonewasslightlyeffective(20%),since asuboptimaltreatmentwasused.Survivalincreasedto60%withOseltamivirandto100%withOseltamivirandNACusedincombination.Since NACalonedoes notshowanyantiviralaction,thepresentfindingssuggestthatantioxidanttherapyin~reasesurvivalby animprovementinhostdefensemechanisms,and/orby adirectantioxidanteffectagainstoxidativestressassociatedwithviralinfection.Ourstudiesdemonstratetheeffectivenessofcombiningagentsactingthroughdifferentmechanisms,suchasantiviraldrugsoseltamivirandtheantioxidantNAC,indicatinga possibleadvantageofcombiningthetwotreatments.Various mediators contribute to the pathogenesisofpulmonary inflammation induced by infectiousagents. In particular, cytokines, chemokines andreactive oxygen species (ROS) have been implicated.Cytokines and chemokines, which are both producedas partofthe host immune response tobacteria(1-2), contribute to the pathogenesisoftissue damage(3-4). ROS contributes to the antibacterial response,evenifoverproductioncan result in oxidative stressthat amplifies the inflammatory response.Many studies have shown that oxidative stress isimportant in the pathog

N-ACETYLCYSTEINE NAC

ZAMBON 3 PAVOS FLUMIL FORTE O TIENDAS DE SUPLES

LA droja SECRETA QUE LOS ILLUMINATI DE LA OMS NO QUIEREN QUE CONOZCA Y QUE USAN ELLOS PARA EMBICHARTE A TI

Tú estás hablando del MMS, aquí están hablando de ESTO:

Fluimucil Forte de 600mg para la expectoración de la mucosidad | Fluimucil

BRUTAL HEALING


N-acetyl-L-cysteine (NAC) inhibits bichito replication and expression of pro-inflammatory molecules in A549 cells infected with highly pathogenic H5N... - PubMed - NCBI

SAGE Journals: Your gateway to world-class research journals

y no vais a creer lo que paso...

Spoiler: allseeyingeye, post: 27936910, member: 6541

NAC ACETIL CISTENIA



THE bichito KILLER


0.20 SEGUNDOS DE INVESTIGACION SUPREMA



1 SHOT


1 KILL



Result Filters saltar el zurrullo este. es solo para que los normies vean que es "cientifico"


Format: Abstract

Biochem Pharmacol. 2010 Feb 1;79(3):413-20. doi: 10.1016/j.bcp.2009.08.025. Epub 2009 Sep 2.

N-acetyl-L-cysteine (NAC) inhibits bichito replication and expression of pro-inflammatory molecules in A549 cells infected with highly pathogenic H5N1 influenza A bichito.

Geiler J1, Michaelis M, Naczk P, Leutz A, Langer K, Doerr HW, Cinatl J Jr.

Author information


Abstract

The antioxidant N-acetyl-L-cysteine (NAC) had been shown to inhibit replication of seasonal human influenza A viruses. Here, the effects of NAC on bichito replication, bichito-induced pro-inflammatory responses and bichito-induced apoptosis were investigated in H5N1-infected lung epithelial (A549) cells. NAC at concentrations ranging from 5 to 15 mM reduced H5N1-induced cytopathogenic effects (CPEs), bichito-induced apoptosis and infectious viral yields 24 h post-infection. NAC also decreased the production of pro-inflammatory molecules (CXCL8, CXCL10, CCL5 and interleukin-6 (IL-6)) in H5N1-infected A549 cells and reduced monocyte migration towards supernatants of H5N1-infected A549 cells. The antiviral and anti-inflammatory mechanisms of NAC included inhibition of activation of oxidant sensitive pathways includ

SAGE Journals: Your gateway to world-class research journals

ERNATIONALJOURNALOFIMMUNOPATHOLOGYANDPHARMACOLOGYVol. 20, no. 2,349-354(2007)N-ACETYLCYSTEINESYNERGIZESWITHOSELTAMIVIRINPROTECTINGMICEFROMLETHALINFLUENZAINFECTIONA. GAROZZO, G. TEMPERA, D.UNGHERI',R.TIMPANARO and A. CASTRODepartmentofMicrobiologicalandGynaecologicalSciences, UniversityofCatania, Catania;JGlobalScientificProjectManagement, Zambon Italia Sirl., Milano, ItalyReceivedSeptember20,2006-AcceptedFebruary20,2007Manystudieshaveshownthatoxidativestressisimportantinthepathogenesisofpulmonarydamageduringinfluenzavirusinfections.Antioxidantmoleculesarethereforepotentiallyusefulagainstviralinfection.OurpreviousstudiesshowthatN-acetytcysteine(NAC)hasa protectiveeffect in amodeloflethalinfluenzainfectionin mice. NACadministrationsignificantlydecreasedthemortalityininfectedmice.FurtherstudieshavedemonstratedthatNACenhancedsurvivalincombinationwiththeantiviralagentribavirin.Inthepresentstudy,wereporttheeffectofcombinedtreatmentwithNACandOseltamivir,clinicallyused inthetreatmentandpreventionofinfluenzavirusinfection,in amurinemodeloflethalinfluenzainfection.NAC was given as a singledailydose of 1000mglKgstartingfrom4 hbeforeinfectionanduntilday4afterinfection;Oseltamivirwas given twicedailyat dose of 1mglKgldiefor 5days,startingfrom4 hbeforeinfection.End-pointevaluationwas21-days'survival.NACalonewasslightlyeffective(20%),since asuboptimaltreatmentwasused.Survivalincreasedto60%withOseltamivirandto100%withOseltamivirandNACusedincombination.Since NACalonedoes notshowanyantiviralaction,thepresentfindingssuggestthatantioxidanttherapyin~reasesurvivalby animprovementinhostdefensemechanisms,and/orby adirectantioxidanteffectagainstoxidativestressassociatedwithviralinfection.Ourstudiesdemonstratetheeffectivenessofcombiningagentsactingthroughdifferentmechanisms,suchasantiviraldrugsoseltamivirandtheantioxidantNAC,indicatinga possibleadvantageofcombiningthetwotreatments.Various mediators contribute to the pathogenesisofpulmonary inflammation induced by infectiousagents. In particular, cytokines, chemokines andreactive oxygen species (ROS) have been implicated.Cytokines and chemokines, which are both producedas partofthe host immune response tobacteria(1-2), contribute to the pathogenesisoftissue damage(3-4). ROS contributes to the antibacterial response,evenifoverproductioncan result in oxidative stressthat amplifies the inflammatory response.Many studies have shown that oxidative stress isimportant in the pathog

No entiendo nada...


(El perrito está genial).

KEK SALVA


NAC SALVA


no se serio, mata bichito influezia de esos


god shit

es que acabo de tomarlo porq ue lo compro a kgs (bueno no, de 200 g o asi)

y la verdad que lo acabo de tomara ahora mismo

y he tenido un ERECCION DEL GLUTATION



Resultado de imagen de nac cistenina






TOP SECRET
BRUTAL HEALINGPROCEDURES
ONLY FOR YOUR EYES
NOT FOR NORMIES EYES

600 × 400

 

[allseeyingeye]

SIEMPRE EN VANGUARDIA

boom!


YA VEREIS

INTERES = ZERO








los multinicks quieren drama y hype

 

[Sargento Kowalski]

Spoiler: allseeyingeye, post: 27936910, member: 6541

NAC ACETIL CISTENIA


THE bichito KILLER

0.20 SEGUNDOS DE INVESTIGACION SUPREMA


1 SHOT

1 KILL

• Advanced
• Help

Result Filters

• Format: Abstract

Send to








Biochem Pharmacol. 2010 Feb 1;79(3):413-20. doi: 10.1016/j.bcp.2009.08.025. Epub 2009 Sep 2.
N-acetyl-L-cysteine (NAC) inhibits bichito replication and expression of pro-inflammatory molecules in A549 cells infected with highly pathogenic H5N1 influenza A bichito.
Geiler J1, Michaelis M, Naczk P, Leutz A, Langer K, Doerr HW, Cinatl J Jr.
Author information

Abstract
The antioxidant N-acetyl-L-cysteine (NAC) had been shown to inhibit replication of seasonal human influenza A viruses. Here, the effects of NAC on bichito replication, bichito-induced pro-inflammatory responses and bichito-induced apoptosis were investigated in H5N1-infected lung epithelial (A549) cells. NAC at concentrations ranging from 5 to 15 mM reduced H5N1-induced cytopathogenic effects (CPEs), bichito-induced apoptosis and infectious viral yields 24 h post-infection. NAC also decreased the production of pro-inflammatory molecules (CXCL8, CXCL10, CCL5 and interleukin-6 (IL-6)) in H5N1-infected A549 cells and reduced monocyte migration towards supernatants of H5N1-infected A549 cells. The antiviral and anti-inflammatory mechanisms of NAC included inhibition of activation of oxidant sensitive pathways includ
SAGE Journals: Your gateway to world-class research journals
ERNATIONALJOURNALOFIMMUNOPATHOLOGYANDPHARMACOLOGYVol. 20, no. 2,349-354(2007)N-ACETYLCYSTEINESYNERGIZESWITHOSELTAMIVIRINPROTECTINGMICEFROMLETHALINFLUENZAINFECTIONA. GAROZZO, G. TEMPERA, D.UNGHERI',R.TIMPANARO and A. CASTRODepartmentofMicrobiologicalandGynaecologicalSciences, UniversityofCatania, Catania;JGlobalScientificProjectManagement, Zambon Italia Sirl., Milano, ItalyReceivedSeptember20,2006-AcceptedFebruary20,2007Manystudieshaveshownthatoxidativestressisimportantinthepathogenesisofpulmonarydamageduringinfluenzavirusinfections.Antioxidantmoleculesarethereforepotentiallyusefulagainstviralinfection.OurpreviousstudiesshowthatN-acetytcysteine(NAC)hasa protectiveeffect in amodeloflethalinfluenzainfectionin mice. NACadministrationsignificantlydecreasedthemortalityininfectedmice.FurtherstudieshavedemonstratedthatNACenhancedsurvivalincombinationwiththeantiviralagentribavirin.Inthepresentstudy,wereporttheeffectofcombinedtreatmentwithNACandOseltamivir,clinicallyused inthetreatmentandpreventionofinfluenzavirusinfection,in amurinemodeloflethalinfluenzainfection.NAC was given as a singledailydose of 1000mglKgstartingfrom4 hbeforeinfectionanduntilday4afterinfection;Oseltamivirwas given twicedailyat dose of 1mglKgldiefor 5days,startingfrom4 hbeforeinfection.End-pointevaluationwas21-days'survival.NACalonewasslightlyeffective(20%),since asuboptimaltreatmentwasused.Survivalincreasedto60%withOseltamivirandto100%withOseltamivirandNACusedincombination.Since NACalonedoes notshowanyantiviralaction,thepresentfindingssuggestthatantioxidanttherapyin~reasesurvivalby animprovementinhostdefensemechanisms,and/orby adirectantioxidanteffectagainstoxidativestressassociatedwithviralinfection.Ourstudiesdemonstratetheeffectivenessofcombiningagentsactingthroughdifferentmechanisms,suchasantiviraldrugsoseltamivirandtheantioxidantNAC,indicatinga possibleadvantageofcombiningthetwotreatments.Various mediators contribute to the pathogenesisofpulmonary inflammation induced by infectiousagents. In particular, cytokines, chemokines andreactive oxygen species (ROS) have been implicated.Cytokines and chemokines, which are both producedas partofthe host immune response tobacteria(1-2), contribute to the pathogenesisoftissue damage(3-4). ROS contributes to the antibacterial response,evenifoverproductioncan result in oxidative stressthat amplifies the inflammatory response.Many studies have shown that oxidative stress isimportant in the pathog

No entiendo nada...

(El perrito está genial).

Edito: parece que dice que esta sustancia EXPECTORANTE funciona contra ese bichito tan famoso,

Acetilcisteína - Wikipedia
N-acetilcisteína: despreciada para el resfriado, ahora regresa como anticancerígena

La N-acetilcisteína (abreviado como NAC) es un fármaco con propiedades mucolíticas. Su mecanismo de acción es romper los enlaces de disulfuro tanto de las secreciones mucosas como de las mucopurulentas, logrando que sean menos viscosas (efecto mucolítico). Este efecto se concentra sobre todo en la disminución de la viscosidad de las secreciones bronquiales, haciendo que sea más fácil la posterior expulsión. También activa el epitelio ciliado, favoreciendo la expectoración y es citoprotector del aparato respiratorio. Es precursor de glutatión, normalizando sus niveles. Sirve para expulsar flema.

 

[Fandiño]

Spoiler: allseeyingeye, post: 27936910, member: 6541

NAC ACETIL CISTENIA


THE bichito KILLER

0.20 SEGUNDOS DE INVESTIGACION SUPREMA


1 SHOT

1 KILL

• Advanced
• Help

Result Filters

• Format: Abstract

Send to








Biochem Pharmacol. 2010 Feb 1;79(3):413-20. doi: 10.1016/j.bcp.2009.08.025. Epub 2009 Sep 2.
N-acetyl-L-cysteine (NAC) inhibits bichito replication and expression of pro-inflammatory molecules in A549 cells infected with highly pathogenic H5N1 influenza A bichito.
Geiler J1, Michaelis M, Naczk P, Leutz A, Langer K, Doerr HW, Cinatl J Jr.
Author information

Abstract
The antioxidant N-acetyl-L-cysteine (NAC) had been shown to inhibit replication of seasonal human influenza A viruses. Here, the effects of NAC on bichito replication, bichito-induced pro-inflammatory responses and bichito-induced apoptosis were investigated in H5N1-infected lung epithelial (A549) cells. NAC at concentrations ranging from 5 to 15 mM reduced H5N1-induced cytopathogenic effects (CPEs), bichito-induced apoptosis and infectious viral yields 24 h post-infection. NAC also decreased the production of pro-inflammatory molecules (CXCL8, CXCL10, CCL5 and interleukin-6 (IL-6)) in H5N1-infected A549 cells and reduced monocyte migration towards supernatants of H5N1-infected A549 cells. The antiviral and anti-inflammatory mechanisms of NAC included inhibition of activation of oxidant sensitive pathways includ
SAGE Journals: Your gateway to world-class research journals
ERNATIONALJOURNALOFIMMUNOPATHOLOGYANDPHARMACOLOGYVol. 20, no. 2,349-354(2007)N-ACETYLCYSTEINESYNERGIZESWITHOSELTAMIVIRINPROTECTINGMICEFROMLETHALINFLUENZAINFECTIONA. GAROZZO, G. TEMPERA, D.UNGHERI',R.TIMPANARO and A. CASTRODepartmentofMicrobiologicalandGynaecologicalSciences, UniversityofCatania, Catania;JGlobalScientificProjectManagement, Zambon Italia Sirl., Milano, ItalyReceivedSeptember20,2006-AcceptedFebruary20,2007Manystudieshaveshownthatoxidativestressisimportantinthepathogenesisofpulmonarydamageduringinfluenzavirusinfections.Antioxidantmoleculesarethereforepotentiallyusefulagainstviralinfection.OurpreviousstudiesshowthatN-acetytcysteine(NAC)hasa protectiveeffect in amodeloflethalinfluenzainfectionin mice. NACadministrationsignificantlydecreasedthemortalityininfectedmice.FurtherstudieshavedemonstratedthatNACenhancedsurvivalincombinationwiththeantiviralagentribavirin.Inthepresentstudy,wereporttheeffectofcombinedtreatmentwithNACandOseltamivir,clinicallyused inthetreatmentandpreventionofinfluenzavirusinfection,in amurinemodeloflethalinfluenzainfection.NAC was given as a singledailydose of 1000mglKgstartingfrom4 hbeforeinfectionanduntilday4afterinfection;Oseltamivirwas given twicedailyat dose of 1mglKgldiefor 5days,startingfrom4 hbeforeinfection.End-pointevaluationwas21-days'survival.NACalonewasslightlyeffective(20%),since asuboptimaltreatmentwasused.Survivalincreasedto60%withOseltamivirandto100%withOseltamivirandNACusedincombination.Since NACalonedoes notshowanyantiviralaction,thepresentfindingssuggestthatantioxidanttherapyin~reasesurvivalby animprovementinhostdefensemechanisms,and/orby adirectantioxidanteffectagainstoxidativestressassociatedwithviralinfection.Ourstudiesdemonstratetheeffectivenessofcombiningagentsactingthroughdifferentmechanisms,suchasantiviraldrugsoseltamivirandtheantioxidantNAC,indicatinga possibleadvantageofcombiningthetwotreatments.Various mediators contribute to the pathogenesisofpulmonary inflammation induced by infectiousagents. In particular, cytokines, chemokines andreactive oxygen species (ROS) have been implicated.Cytokines and chemokines, which are both producedas partofthe host immune response tobacteria(1-2), contribute to the pathogenesisoftissue damage(3-4). ROS contributes to the antibacterial response,evenifoverproductioncan result in oxidative stressthat amplifies the inflammatory response.Many studies have shown that oxidative stress isimportant in the pathog

No entiendo nada...

(El perrito está genial).

Yo tampoco.

 

[allseeyingeye]

y no vais a creer lo que paso...

Spoiler: allseeyingeye, post: 27936910, member: 6541

NAC ACETIL CISTENIA


THE bichito KILLER

0.20 SEGUNDOS DE INVESTIGACION SUPREMA


1 SHOT

1 KILL


Result Filters saltar el zurrullo este. es solo para que los normies vean que es "cientifico"

• Format: Abstract

Biochem Pharmacol. 2010 Feb 1;79(3):413-20. doi: 10.1016/j.bcp.2009.08.025. Epub 2009 Sep 2.
N-acetyl-L-cysteine (NAC) inhibits bichito replication and expression of pro-inflammatory molecules in A549 cells infected with highly pathogenic H5N1 influenza A bichito.
Geiler J1, Michaelis M, Naczk P, Leutz A, Langer K, Doerr HW, Cinatl J Jr.
Author information

Abstract
The antioxidant N-acetyl-L-cysteine (NAC) had been shown to inhibit replication of seasonal human influenza A viruses. Here, the effects of NAC on bichito replication, bichito-induced pro-inflammatory responses and bichito-induced apoptosis were investigated in H5N1-infected lung epithelial (A549) cells. NAC at concentrations ranging from 5 to 15 mM reduced H5N1-induced cytopathogenic effects (CPEs), bichito-induced apoptosis and infectious viral yields 24 h post-infection. NAC also decreased the production of pro-inflammatory molecules (CXCL8, CXCL10, CCL5 and interleukin-6 (IL-6)) in H5N1-infected A549 cells and reduced monocyte migration towards supernatants of H5N1-infected A549 cells. The antiviral and anti-inflammatory mechanisms of NAC included inhibition of activation of oxidant sensitive pathways includ
SAGE Journals: Your gateway to world-class research journals
ERNATIONALJOURNALOFIMMUNOPATHOLOGYANDPHARMACOLOGYVol. 20, no. 2,349-354(2007)N-ACETYLCYSTEINESYNERGIZESWITHOSELTAMIVIRINPROTECTINGMICEFROMLETHALINFLUENZAINFECTIONA. GAROZZO, G. TEMPERA, D.UNGHERI',R.TIMPANARO and A. CASTRODepartmentofMicrobiologicalandGynaecologicalSciences, UniversityofCatania, Catania;JGlobalScientificProjectManagement, Zambon Italia Sirl., Milano, ItalyReceivedSeptember20,2006-AcceptedFebruary20,2007Manystudieshaveshownthatoxidativestressisimportantinthepathogenesisofpulmonarydamageduringinfluenzavirusinfections.Antioxidantmoleculesarethereforepotentiallyusefulagainstviralinfection.OurpreviousstudiesshowthatN-acetytcysteine(NAC)hasa protectiveeffect in amodeloflethalinfluenzainfectionin mice. NACadministrationsignificantlydecreasedthemortalityininfectedmice.FurtherstudieshavedemonstratedthatNACenhancedsurvivalincombinationwiththeantiviralagentribavirin.Inthepresentstudy,wereporttheeffectofcombinedtreatmentwithNACandOseltamivir,clinicallyused inthetreatmentandpreventionofinfluenzavirusinfection,in amurinemodeloflethalinfluenzainfection.NAC was given as a singledailydose of 1000mglKgstartingfrom4 hbeforeinfectionanduntilday4afterinfection;Oseltamivirwas given twicedailyat dose of 1mglKgldiefor 5days,startingfrom4 hbeforeinfection.End-pointevaluationwas21-days'survival.NACalonewasslightlyeffective(20%),since asuboptimaltreatmentwasused.Survivalincreasedto60%withOseltamivirandto100%withOseltamivirandNACusedincombination.Since NACalonedoes notshowanyantiviralaction,thepresentfindingssuggestthatantioxidanttherapyin~reasesurvivalby animprovementinhostdefensemechanisms,and/orby adirectantioxidanteffectagainstoxidativestressassociatedwithviralinfection.Ourstudiesdemonstratetheeffectivenessofcombiningagentsactingthroughdifferentmechanisms,suchasantiviraldrugsoseltamivirandtheantioxidantNAC,indicatinga possibleadvantageofcombiningthetwotreatments.Various mediators contribute to the pathogenesisofpulmonary inflammation induced by infectiousagents. In particular, cytokines, chemokines andreactive oxygen species (ROS) have been implicated.Cytokines and chemokines, which are both producedas partofthe host immune response tobacteria(1-2), contribute to the pathogenesisoftissue damage(3-4). ROS contributes to the antibacterial response,evenifoverproductioncan result in oxidative stressthat amplifies the inflammatory response.Many studies have shown that oxidative stress isimportant in the pathog

No entiendo nada...

(El perrito está genial).

KEK SALVA

NAC SALVA

no se serio, mata bichito influezia de esos

god shit
es que acabo de tomarlo porq ue lo compro a kgs (bueno no, de 200 g o asi)
y la verdad que lo acabo de tomara ahora mismo
y he tenido un ERECCION DEL GLUTATION

 

[Sargento Kowalski]

Ésa es la más cara. ¿Tiene algo de especial?

Ésta está bien de precio:

 

[allseeyingeye]

Ésa es la más cara. ¿Tiene algo de especial?

NADA seguramente
pillo de myprotein y de ... jorobar como era.,, bulkpowders

en la farmacia vale , no se, creo que 3 pavos con sabor limon 20 dosis creo
yo creo que con eso resulto

FLUMIL FORTE 600 mg creo
limon ta bien

 

[allseeyingeye]

N-ACETYLCYSTEINE


SAGE Journals: Your gateway to world-class research journals

SAGE Journals: Your gateway to world-class research journals

 

[allseeyingeye]

N-ACETYLCYSTEINE NAC

ZAMBON 3 PAVOS FLUMIL FORTE O TIENDAS DE SUPLES

LA droja SECRETA QUE LOS ILLUMINATI DE LA OMS NO QUIEREN QUE CONOZCAS Y QUE USAN ELLOS PARA EMBICHARTE A TI


BRUTAL HEALING


N-acetyl-L-cysteine (NAC) inhibits bichito replication and expression of pro-inflammatory molecules in A549 cells infected with highly pathogenic H5N... - PubMed - NCBI

SAGE Journals: Your gateway to world-class research journals

TOP SECRET
BRUTAL HEALINGPROCEDURES
ONLY FOR YOUR EYES
NOT FOR NORMIES EYES

600 × 400

 

[kerri]

pero es en serio o es en coña.
pregunto en serio.

 

[elmegaduque]

¿Y no será mejor beberse medio litro de lejía?.

En Zaragoza un yonki lo hizo para dar de baja de la suscripción de la vida el sidra.

 

[allseeyingeye]

¿Y no será mejor beberse medio litro de lejía?.

En Zaragoza un yonki lo hizo para dar de baja de la suscripción de la vida el sidra.

a ver cachopo
que es flumil forte jorobar
sabe a naranja o limon

pero claro, veo que eres uno de esos que quiere moricccccccsrsssscc

 

[allseeyingeye]

pero es en serio o es en coña.
pregunto en serio.

totalmente en serio xD

la gracia esta, en que es algo absolutamente VIEJUNO en el sentido de SABIDO

por eso todo el disparate este de la crisis de pollo frito el bichito 19 bla bla bla

 

[Sargento Kowalski]

¿Y no será mejor beberse medio litro de lejía?.

En Zaragoza un yonki lo hizo para dar de baja de la suscripción de la vida el sidra.

Tú estás hablando del MMS, aquí están hablando de ESTO:

Fluimucil Forte de 600mg para la expectoración de la mucosidad | Fluimucil

 

[Sargento Kowalski]

Up, por si le sirve a alguien.